Browsing by Author "Smith, David J."
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Item Open Access A model to predict maximal lactate steady state(2007) Neupert, Emma; Smith, David J.Item Open Access A novel physiological model examining the tolerance to exercise above maximal lactate steady state(2012) Groves, Erik M.; Smith, David J.; Reimer, RayleneItem Open Access Characterizing oxygen consumption during severe exhaustive cycling(2001) Hawley, Jodi Ann; Smith, David J.Item Open Access Comparison and reliability of non-invasive acetylene uptake techniques for the measurement of cardiac output(2003) Dibski, Daniel William; Smith, David J.Item Open Access Design and validation of a multi-stage skating specific test to predict aerobic power in competivie figure skaters(1999) Zapalo, Peter Paul III; Smith, David J.Item Open Access Does maximal strength training improve the efficiency and sustainability of performance in trained male cyclists?(2011) Aylwin, Anna J. M.; Smith, David J.Item Open Access Fluctuations in serum iron with high-intensity exercise(1987) Roberts, Delia (Delia Beth), 1957-; Smith, David J.Much work has been done on the chronic effects of exercise and the development of iron deficiency. However, few investigators have assessed the acute effects of exercise on serum iron. A series of experiments were designed to isolate and evaluate the effects of short- duration, high-intensity exercise. Subjects were male physical education students, well-trained biathletes and elite speedskaters. The exercise stimulus was 1 or 2 30-second supramaximal bouts on a cycle ergometer. Physical education students showed one of two distinct responses to the exercise. Group A demonstrated an increase in serum Fe and a decrease in α₁AT. Group B demonstrated an acute inflammatory 1 response to the same exercise with a decrease in serum Fe and an increase in α₁AT. The correlation coefficient for these two variables for experiments 3-7 inclusive was -0.83. No difference was seen between the two groups for the measures of glycolytic capacity, mean power output over 30 seconds and peak blood lactate concentration. Well- trained and elite athletes showed a tendency toward the group A response. However, an aerobic training program carried out by physical education students increased aerobic capacity by 15%, without affecting the serum Fe response to high-intensity exercise. A short-term anaerobic training program which did not affect mean power output or peak blood lactate, did produce a shift of subjects showing a group B response to a group A response. The results suggest that there is a biochemical response to exercise indicative of inflammation which is altered by high-intensity training, but which is also distinct from existing measures of anaerobic and aerobic capacity. This response is suggested to be affected by the total volume of stress exerted upon an individual at any given time. Elicitation of an inflammatory response may thus depend upon the tolerance of an individual to stress, as determined by previous exposures to stress.Item Open Access The effect of a prescribed increase in training intensity on plasma glutamine and glutamate concentration and performance(1999) Henderson, Jennifer; Smith, David J.Item Open Access The effect of acute heat exposure and handcooling on power output during ballistic strength training(2010) Challis, Graeme Grant; Smith, David J.; Kolb, JonItem Open Access The effect of high-intensity interval training on stroke volume and endurance performance in trained endurance athletes(2005) McGillivray, Meegan Anne; Smith, David J.This study compared the effects of two training protocols, high intensity (HIT) and low intensity training (LIT) on cardiovascular and performance adaptations. Twelve male cyclists (V02 max 61.7±3.6 mL·ki1 ·min-1) completed an HIT or LIT, volume-matched periodized training plan. Maximum stroke volume increased for all subjects following long duration training and decreased with HIT and LIT (p=0.002). V0 max and 2 maximum cardiac output did not change. Lactate minimum power output increased 5% following duration training, and then a further 9% (275 to 297W) with HIT but not LIT. Peak aerobic power output increased from 425W to 460W with HIT, 405W to 425W for the LIT group with taper. Time trial performance increased linearly for both groups (p < 0.001). The data demonstrates that HIT does not change maximal cardiac output when following long duration training, and time trial perItem Open Access The effects of normabaric hypoxia on erythropoietin production and exercise performance(1997) Simard, Stephane; Smith, David J.Item Open Access Validation of a segmented model of critical speed and its dominant physiological componenets(1998) Jacobson, Perry; Smith, David J.